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The patients;, aged 30 to 56, had HD of 7 to 12 years’ duration. Their condition has been slowly progressive and unresponsive to prior therapy with neuroleptics. Orally administered CBD was initiated at 300 mg/d and increased 1 week later to 600 mg/d for the next 3 weeks. toxicity in acute studies (Hollister, 1973; Carlini et al, 1979). which 8 epileptic patients received 200 or 300 mg for up to 4 1/2 months.

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Based on anecdotal reports of improvement of generalized dystonia with marijuana smoking, we decided to try CBD in a patient with severe cranial dystonia . At 300 to 400 mg/d, there was no further improvement in the dyskinesia, and adverse effects appeared.

Neurological examinations of all other patients were within normal limits. Withdrawal of CBD for 24 hours resulted in reappearance of severe spasm at 25 to 30 per min. Side effects included dry mouth, transient morning headache, and slight sedation. CBD was initiated at 100 mg/day, in divided doses, and slowly increased over several weeks to 400 mg/day. Spasm frequency, counted twice daily by a relative while the patient was either talking or driving, gradually decreased from 20 to 30 per min before CBD to 7 or 15 per min at a dosage of 400 mg/day.

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In idiopathic dystonia, the terapeutic effect of marijuana smoking is reported to be comparable to diazepam (C.D. Marsden, in Disorders of Movement, 1981,81). Cannabidiol has been shown to have significant muscle relaxant effects and to reduce muscular spasms in humans .

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Following CBD administration, he was able to walk without support, an effect that lasted about 24 hours. We have therefore tested the efficacy of CBD in two patients with dystonic movement disorders. Oral doses of CBD rising from 100 to 600 mg/day over a 6 week period were where to buy cbd gummies near me administered along with standard medication.